Production of TNF-.ALPHA. by mononuclear cells and urinary TNF-.ALPHA. in glomerulopthies of childhood.
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منابع مشابه
TNF-alpha in asthma.
Although only 5-10% of patients with asthma are relatively unresponsive to treatment with inhaled corticosteroids, refractory asthma represents an important condition, as these patients suffer considerable morbidity and mortality and consume a disproportionately large amount of health resource. Treatment options are limited and there is a large unmet clinical need for additional therapies. Tumo...
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ed by: African Index Medicus (WHO), CAB Abstracts, Global Health Abstracts, Asian Science Index, Index Veterinarius Full-text available at http://www.ajbrui.com & http://www.bioline.br/md Received: January, 2006 Accepted (Revised): April, 2006 Published May, 2006 Full Length Research Article Cardiospermum halicacabum suppresses the production of TNF-alpha and Nitric oxide by Human Peripheral Bl...
متن کاملThe Possible Role of TNF-alpha in Physiological and Pathophysiological Cardiac Hypertrophy in Rats
Pathological cardiac hypertrophy was produced by partial abdominal aortic constriction (PAAC) for 4 wk, while physiological cardiac hypertrophy was produced by chronic swimming training (CST) for 8 wk in rats. Pentoxifylline (30 mg/kg, 300 mg/kg i.p., day-1) treatment was started three days before PAAC and CST and it was continued for 4 wk in PAAC and 8 wk in CST experimental model. The left ve...
متن کاملEffects of taxol on TNF-alpha and IL-6 production by human peripheral blood cells.
Taxol, a new drug isolated from the bark of the Pacific yew tree, is being used in treating patients with breast, ovary, lung, neck, and brain cancer.’.* The therapeutic mechanisms of taxol are not yet completely known. Taxol binds to p-tubulin, stabilizes microtubules against dep~lymerization,~ and interferes with microtubules during the cell divisions, blocking the cell through me tapha~e .~ ...
متن کاملDiethyldithiocarbamate enhances production of nitric oxide and TNF-alpha by lipopolysaccharide-stimulated rat Kupffer cells.
Previous studies have shown that large doses of diethyldithiocarbamate (DDC) cause liver injury in rats and the pathogenesis of this injury involves, in part, release of superoxide anion by Kupffer cells. The purpose of this study was to evaluate if DDC was able to stimulate other potentially toxic mediators such as nitric oxide (NO) and tumor necrosis factor-alpha (TNF-alpha) using isolated ra...
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ژورنال
عنوان ژورنال: Nihon Shoni Jinzobyo Gakkai Zasshi
سال: 1997
ISSN: 1881-3933,0915-2245
DOI: 10.3165/jjpn.10.133